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“Colon Cancer That Runs in Families” (Familial Adenomatous Polyposis), APC Gene, Symptoms, Treatment
“Colon Cancer That Runs in Families” (Familial Adenomatous Polyposis), APC Gene, Symptoms, Diagnosis, Treatment
Familial Adenomatous Polyposis is an autosomal dominant inherited condition involving significantly increased risk of colorectal or colon cancer. Familial Adenomatous Polyposis can lead to colon cancer very early on in life, roughly by the age of 40 in most individuals. Familial Adenomatous Polyposis is caused by a mutation in the APC gene, which itself is involved in a biochemical pathway known as the Wnt-B catenin signaling pathway. Familial Adenomatous Polyposis often has no symptoms until the onset of colorectal cancer. In this lesson, we discuss the pathophysiology behind with Familial Adenomatous Polyposis occurs, how it is diagnosed and how it is treated.
I hope you find...
published: 27 Jan 2022
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Familial Adenomatous Polyposis (FAP) | More than 100 polyps in my colon 😱 | Colon Cancer
Familial Adenomatous Polyposis (FAP) | Genetic disease…More than 100 polyps in my colon 😱 | About 100% Chance of Developing Colorectal Carcinoma…Elective Colectomy is often performed.
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published: 06 Apr 2023
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Familial Adenomatous Polyposis ( FAP): Everything You Need To Know
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Chapters
0:00 Introduction
0:56 Symptoms of Familial Adenomatous Polyposis
2:20 Diagnosis for Familial Adenomatous Polyposis
2:50 Treatment for Familial Adenomatous Polyposis
Familial adenomatous polyposis (FAP) is an autosomal dominant inherited condition in which numerous adenomatous polyps form mainly in the epithelium of the large intestine. While these polyps start out benign, malignant transformation into colon cancer occurs when they are left untreated. Three variants are known to exist, FAP and attenuated FAP (originally called hereditary flat adenoma syndrome[1]) are caused by APC gene defects on chromosome 5 while autosomal recessive FAP (or MUTYH-associated polyposis) is caused by defects in the MUTYH gene on chromosome 1. Of the three, FAP itself is the most severe a...
published: 09 Sep 2022
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FAP vs Lynch (Familial Adenomatous Polyposis vs Hereditary Nonpolyposis Colorectal cancer)| Oncology
FAP vs Lynch (Familial Adenomatous Polyposis vs Hereditary Nonpolyposis Colorectal cancer)| Oncology | Pathology | 5-Minute-Review.
Familial adenomatous polyposis is a disease characterized by the presence of multiple polyps in the colon. It carries very high risk of progressing into colon cancer (colorectal carcinoma). On the other hand, hereditary nonpolyposis colorectal cancer (Lynch syndrome) doesn’t have many polyps in the colon, it’s a colorectal cancer.
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published: 16 Apr 2023
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Colon: Familial Adenomatous Polyposis
Gross and microscopic description of adenomatous polyps in familial adenomatous polyposis.
Video source: Pathweb
Register for FREE access to Pathweb: https://nus.syd1.qualtrics.com/jfe/form/SV_aeYDYT8bvIP4Ii9
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Pathweb instagram: https://www.instagram.com/pathweb/
published: 27 Nov 2019
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What is Familial Adenomatous Polyposis or FAP?
From ASCO 2011 -- A discussion with Eric Charsky, disabled veteran, cancer survivor, and Founder and CEO of Volontie 1.
published: 27 Jun 2011
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Laparoscopic Assisted Restorative Proctocolectomy for Familial Adenomatous Polyposis
Authors: Steven D. Wexner, MD, FACS, FRCS, FRCS (Ed); Jill C. Genua, MD; David A. Vivas, MD
published: 27 Jun 2011
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Familial Adenomatous Polyposis (FAP)
Watch the video to know about FAP and its associated syndromes.
published: 04 Mar 2016
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Gastric adenomas and their management in familial adenomatous polyposis
Isabel Martin on "Gastric adenomas and their management in familial adenomatous polyposis"
Authors: Isabel Martin, Victorine H. Roos, Chukwuemeka Anele et al.
Summary:
Among 726 patients with familial adenomatous polyposis (FAP), 104 (14%) had a gastric adenoma. High grade dysplasia was detected in five (5%) of these patients, while two patients had gastric cancer at initial gastric adenoma diagnosis. During follow-up, three patients were additionally diagnosed with gastric cancer. The study shows the importance of gastric endoscopic surveillance in patients with FAP.
Bibliography
Endoscopy 2021; 53: 795–801
DOI 10.1055/a-1265-2716
ISSN 0013-726X
© 2020. Thieme. All rights reserved.
Georg Thieme Verlag KG, Rüdigerstraße 14,
70469 Stuttgart, Germany
Read more here: https://doi.org/10.10...
published: 27 Jul 2021
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Attenuated- Familial Adenomatous Polyposis
Attenuated (Mild Type) Familial Adenomatous Polyposis
Colorectal cancers usually develop from polyps. If there is an adenomatous polyp, that means there is a risk of cancer. This is bad news. The good news is that removing polyps eliminates the risk of cancer.
Polyps are best diagnosed with colonoscopy and can be removed with the help of colonoscopy. If there are too many polyps that cannot be removed by colonoscopy, the large intestine including the polyps should be surgically removed.
Some genetic diseases can cause huge numbers of polyps in the large intestine. These genetic diseases pass from the parents to their children.
A genetic disease that causes more than 100 polyps in the large intestine is called familial polyposis. There is also a mild type of this disease. In the mild ...
published: 12 Sep 2020
9:02
“Colon Cancer That Runs in Families” (Familial Adenomatous Polyposis), APC Gene, Symptoms, Treatment
“Colon Cancer That Runs in Families” (Familial Adenomatous Polyposis), APC Gene, Symptoms, Diagnosis, Treatment
Familial Adenomatous Polyposis is an autosomal ...
“Colon Cancer That Runs in Families” (Familial Adenomatous Polyposis), APC Gene, Symptoms, Diagnosis, Treatment
Familial Adenomatous Polyposis is an autosomal dominant inherited condition involving significantly increased risk of colorectal or colon cancer. Familial Adenomatous Polyposis can lead to colon cancer very early on in life, roughly by the age of 40 in most individuals. Familial Adenomatous Polyposis is caused by a mutation in the APC gene, which itself is involved in a biochemical pathway known as the Wnt-B catenin signaling pathway. Familial Adenomatous Polyposis often has no symptoms until the onset of colorectal cancer. In this lesson, we discuss the pathophysiology behind with Familial Adenomatous Polyposis occurs, how it is diagnosed and how it is treated.
I hope you find this lesson helpful. If you do, please like and subscribe for more lessons like this one!
JJ
**MEDICAL LEGAL DISCLAIMER**: JJ Medicine does not provide medical advice, and the information available on this channel does not offer a diagnosis or advice regarding treatment. Information presented in these lessons is for educational purposes ONLY, and information presented here is not to be used as an alternative to a healthcare professional’s diagnosis and treatment of any person/animal. Only a physician or other licensed healthcare professional are able to determine the requirement for medical assistance to be given to a patient. Please seek the advice of your physician or other licensed healthcare provider if you have any questions regarding a medical condition.
*AFFILIATE DISCLAIMER: This YouTube Channel uses affiliate links and may earn a commission from associated sales.
*IMAGE DISCLAIMER: The content (ex. images) used in this lesson are used in accordance with Fair Use laws and are intended for educational/teaching purposes only*
*Subscribe for more free medical lessons* https://www.youtube.com/channel/UCFPvnkCZbHfBvV8ApBBE0vA?sub_confirmation=1
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https://wn.com/“Colon_Cancer_That_Runs_In_Families”_(Familial_Adenomatous_Polyposis),_Apc_Gene,_Symptoms,_Treatment
“Colon Cancer That Runs in Families” (Familial Adenomatous Polyposis), APC Gene, Symptoms, Diagnosis, Treatment
Familial Adenomatous Polyposis is an autosomal dominant inherited condition involving significantly increased risk of colorectal or colon cancer. Familial Adenomatous Polyposis can lead to colon cancer very early on in life, roughly by the age of 40 in most individuals. Familial Adenomatous Polyposis is caused by a mutation in the APC gene, which itself is involved in a biochemical pathway known as the Wnt-B catenin signaling pathway. Familial Adenomatous Polyposis often has no symptoms until the onset of colorectal cancer. In this lesson, we discuss the pathophysiology behind with Familial Adenomatous Polyposis occurs, how it is diagnosed and how it is treated.
I hope you find this lesson helpful. If you do, please like and subscribe for more lessons like this one!
JJ
**MEDICAL LEGAL DISCLAIMER**: JJ Medicine does not provide medical advice, and the information available on this channel does not offer a diagnosis or advice regarding treatment. Information presented in these lessons is for educational purposes ONLY, and information presented here is not to be used as an alternative to a healthcare professional’s diagnosis and treatment of any person/animal. Only a physician or other licensed healthcare professional are able to determine the requirement for medical assistance to be given to a patient. Please seek the advice of your physician or other licensed healthcare provider if you have any questions regarding a medical condition.
*AFFILIATE DISCLAIMER: This YouTube Channel uses affiliate links and may earn a commission from associated sales.
*IMAGE DISCLAIMER: The content (ex. images) used in this lesson are used in accordance with Fair Use laws and are intended for educational/teaching purposes only*
*Subscribe for more free medical lessons* https://www.youtube.com/channel/UCFPvnkCZbHfBvV8ApBBE0vA?sub_confirmation=1
-------------------------------------------------------------------------------------------------------------
For books and more information on these topics
https://www.amazon.com/shop/jjmedicine
Support future lessons and lectures ➜ https://www.patreon.com/jjmedicine
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*Check Out Some of My Other Lessons*
Medical Terminology - The Basics - Lesson 1:
https://www.youtube.com/watch?v=04Wh2E9oNug
Infectious Disease Playlist
https://www.youtube.com/playlist?list=PLRjNoiRtdFwW0Bkc6c66uDACkzotaK26b
Dermatology Playlist
https://www.youtube.com/playlist?list=PLRjNoiRtdFwW3dgORRNmk_MZvnWpZx_lW
Pharmacology Playlist
https://www.youtube.com/playlist?list=PLRjNoiRtdFwWPWcV9lZTh-Qxv0gYakQXR
Hematology Playlist
https://www.youtube.com/playlist?list=PLRjNoiRtdFwW8Ajo1dmxdj4EYEKcbtGju
Rheumatology Playlist
https://www.youtube.com/playlist?list=PLRjNoiRtdFwWDvuhasXsuTFXJp05wwUle
Endocrinology Playlist
https://www.youtube.com/playlist?list=PLRjNoiRtdFwUuRSzhploohnaWVvbVZ7ZP
Nephrology Playlist
https://www.youtube.com/playlist?list=PLRjNoiRtdFwXRSwrFQK5WwWZLk1Las8v8
Fatty Acid Synthesis Pathway:
https://www.youtube.com/watch?v=WuQS_LpNMzo
Wnt/B Catenin Signaling Pathway:
https://www.youtube.com/watch?v=NGVP4J9jpgs
Upper vs. Lower Motor Neuron Lesions:
https://www.youtube.com/watch?v=itNd74V53ng
Lesson on the Purine Synthesis and Salvage Pathway:
https://www.youtube.com/watch?v=e2KFVvI8Akk
Gastrulation | Formation of Germ Layers:
https://www.youtube.com/watch?v=d6Kkn0SECJ4
Introductory lesson on Autophagy (Macroautophagy):
https://www.youtube.com/watch?v=UmSVKzHc5yA
- published: 27 Jan 2022
- views: 37703
7:28
Familial Adenomatous Polyposis (FAP) | More than 100 polyps in my colon 😱 | Colon Cancer
Familial Adenomatous Polyposis (FAP) | Genetic disease…More than 100 polyps in my colon 😱 | About 100% Chance of Developing Colorectal Carcinoma…Elective Colect...
Familial Adenomatous Polyposis (FAP) | Genetic disease…More than 100 polyps in my colon 😱 | About 100% Chance of Developing Colorectal Carcinoma…Elective Colectomy is often performed.
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- published: 06 Apr 2023
- views: 18331
3:58
Familial Adenomatous Polyposis ( FAP): Everything You Need To Know
.
Chapters
0:00 Introduction
0:56 Symptoms of Familial Adenomatous Polyposis
2:20 Diagnosis for Familial Adenomatous Polyposis
2:50 Treatment for Familial A...
.
Chapters
0:00 Introduction
0:56 Symptoms of Familial Adenomatous Polyposis
2:20 Diagnosis for Familial Adenomatous Polyposis
2:50 Treatment for Familial Adenomatous Polyposis
Familial adenomatous polyposis (FAP) is an autosomal dominant inherited condition in which numerous adenomatous polyps form mainly in the epithelium of the large intestine. While these polyps start out benign, malignant transformation into colon cancer occurs when they are left untreated. Three variants are known to exist, FAP and attenuated FAP (originally called hereditary flat adenoma syndrome[1]) are caused by APC gene defects on chromosome 5 while autosomal recessive FAP (or MUTYH-associated polyposis) is caused by defects in the MUTYH gene on chromosome 1. Of the three, FAP itself is the most severe and most common; although for all three, the resulting colonic polyps and cancers are initially confined to the colon wall. Detection and removal before metastasis outside the colon can greatly reduce and in many cases eliminate the spread of cancer.
The root cause of FAP is understood to be a genetic mutation—a change in the body's tumour suppressor genes that prevent development of tumours. The change allows numerous cells of the intestinal wall to develop into potentially cancerous polyps when they would usually reach the end of their life; inevitably one or more will eventually progress and give rise to cancer (7% risk by age 21, rising to 87% by age 45 and 93% by age 50). These gene changes do not trigger cancer, but rather, they reduce the body's ability to prevent cells from becoming cancerous. Even with the gene change, it may still take time before a cell actually does develop that is cancerous as a result, and the gene may in some cases still partially operate to control tumours, therefore cancer from FAP takes many years to develop and is almost always an adult-onset disease.
The second form of FAP, known as attenuated familial adenomatous polyposis has the APC gene functional but slightly impaired. It is therefore somewhat able to operate as usual. Attenuated FAP still presents a high 70% lifetime risk of cancer (as estimated), but typically presents with far fewer polyps (typically 30) rather than the hundreds or thousands usually found in FAP,[2] and arises at an age when FAP is usually no longer considered likely—typically between 40 and 70 years old (average 55[3]) rather than the more usual 30s upward. Because it has far fewer polyps, options for management may be different.[2]
The third variant, autosomal recessive familial adenomatous polyposis or MUTYH-associated polyposis, is also milder and, as its name suggests, requires both parents to be 'carriers' to manifest the condition.
In some cases FAP can manifest higher in the colon than usual (for example, the ascending colon,[citation needed] or proximal to the splenic flexure, or in the stomach or duodenum[1]) where they show no symptoms until cancer is present and greatly advanced. APC mutations have been linked to certain other cancers such as thyroid cancer. As the mutation causing FAP is autosomal dominant, it can be inherited directly from either parent to a child. A genetic blood test of the APC gene exists that can determine whether it is present, and therefore can predict the possibility of FAP. Individuals at risk (due to family links or genetic testing) are usually offered routine monitoring of the intestinal tract every 1–3 years for life, from puberty for FAP and early adulthood for attenuated forms. Colon resection surgery is recommended if numerous colon polyps are found due to high risk of early death from colon cancer. International polyposis registries exist that track known cases of FAP or APC gene defects, for research and clinical purposes. Mutation of APC also occurs commonly in incident cases of colorectal carcinoma, emphasizing its importance in this form of cancer.
https://wn.com/Familial_Adenomatous_Polyposis_(_Fap)_Everything_You_Need_To_Know
.
Chapters
0:00 Introduction
0:56 Symptoms of Familial Adenomatous Polyposis
2:20 Diagnosis for Familial Adenomatous Polyposis
2:50 Treatment for Familial Adenomatous Polyposis
Familial adenomatous polyposis (FAP) is an autosomal dominant inherited condition in which numerous adenomatous polyps form mainly in the epithelium of the large intestine. While these polyps start out benign, malignant transformation into colon cancer occurs when they are left untreated. Three variants are known to exist, FAP and attenuated FAP (originally called hereditary flat adenoma syndrome[1]) are caused by APC gene defects on chromosome 5 while autosomal recessive FAP (or MUTYH-associated polyposis) is caused by defects in the MUTYH gene on chromosome 1. Of the three, FAP itself is the most severe and most common; although for all three, the resulting colonic polyps and cancers are initially confined to the colon wall. Detection and removal before metastasis outside the colon can greatly reduce and in many cases eliminate the spread of cancer.
The root cause of FAP is understood to be a genetic mutation—a change in the body's tumour suppressor genes that prevent development of tumours. The change allows numerous cells of the intestinal wall to develop into potentially cancerous polyps when they would usually reach the end of their life; inevitably one or more will eventually progress and give rise to cancer (7% risk by age 21, rising to 87% by age 45 and 93% by age 50). These gene changes do not trigger cancer, but rather, they reduce the body's ability to prevent cells from becoming cancerous. Even with the gene change, it may still take time before a cell actually does develop that is cancerous as a result, and the gene may in some cases still partially operate to control tumours, therefore cancer from FAP takes many years to develop and is almost always an adult-onset disease.
The second form of FAP, known as attenuated familial adenomatous polyposis has the APC gene functional but slightly impaired. It is therefore somewhat able to operate as usual. Attenuated FAP still presents a high 70% lifetime risk of cancer (as estimated), but typically presents with far fewer polyps (typically 30) rather than the hundreds or thousands usually found in FAP,[2] and arises at an age when FAP is usually no longer considered likely—typically between 40 and 70 years old (average 55[3]) rather than the more usual 30s upward. Because it has far fewer polyps, options for management may be different.[2]
The third variant, autosomal recessive familial adenomatous polyposis or MUTYH-associated polyposis, is also milder and, as its name suggests, requires both parents to be 'carriers' to manifest the condition.
In some cases FAP can manifest higher in the colon than usual (for example, the ascending colon,[citation needed] or proximal to the splenic flexure, or in the stomach or duodenum[1]) where they show no symptoms until cancer is present and greatly advanced. APC mutations have been linked to certain other cancers such as thyroid cancer. As the mutation causing FAP is autosomal dominant, it can be inherited directly from either parent to a child. A genetic blood test of the APC gene exists that can determine whether it is present, and therefore can predict the possibility of FAP. Individuals at risk (due to family links or genetic testing) are usually offered routine monitoring of the intestinal tract every 1–3 years for life, from puberty for FAP and early adulthood for attenuated forms. Colon resection surgery is recommended if numerous colon polyps are found due to high risk of early death from colon cancer. International polyposis registries exist that track known cases of FAP or APC gene defects, for research and clinical purposes. Mutation of APC also occurs commonly in incident cases of colorectal carcinoma, emphasizing its importance in this form of cancer.
- published: 09 Sep 2022
- views: 8013
8:22
FAP vs Lynch (Familial Adenomatous Polyposis vs Hereditary Nonpolyposis Colorectal cancer)| Oncology
FAP vs Lynch (Familial Adenomatous Polyposis vs Hereditary Nonpolyposis Colorectal cancer)| Oncology | Pathology | 5-Minute-Review.
Familial adenomatous polypo...
FAP vs Lynch (Familial Adenomatous Polyposis vs Hereditary Nonpolyposis Colorectal cancer)| Oncology | Pathology | 5-Minute-Review.
Familial adenomatous polyposis is a disease characterized by the presence of multiple polyps in the colon. It carries very high risk of progressing into colon cancer (colorectal carcinoma). On the other hand, hereditary nonpolyposis colorectal cancer (Lynch syndrome) doesn’t have many polyps in the colon, it’s a colorectal cancer.
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https://wn.com/Fap_Vs_Lynch_(Familial_Adenomatous_Polyposis_Vs_Hereditary_Nonpolyposis_Colorectal_Cancer)|_Oncology
FAP vs Lynch (Familial Adenomatous Polyposis vs Hereditary Nonpolyposis Colorectal cancer)| Oncology | Pathology | 5-Minute-Review.
Familial adenomatous polyposis is a disease characterized by the presence of multiple polyps in the colon. It carries very high risk of progressing into colon cancer (colorectal carcinoma). On the other hand, hereditary nonpolyposis colorectal cancer (Lynch syndrome) doesn’t have many polyps in the colon, it’s a colorectal cancer.
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- published: 16 Apr 2023
- views: 7363
5:04
Colon: Familial Adenomatous Polyposis
Gross and microscopic description of adenomatous polyps in familial adenomatous polyposis.
Video source: Pathweb
Register for FREE access to Pathweb: https://...
Gross and microscopic description of adenomatous polyps in familial adenomatous polyposis.
Video source: Pathweb
Register for FREE access to Pathweb: https://nus.syd1.qualtrics.com/jfe/form/SV_aeYDYT8bvIP4Ii9
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https://wn.com/Colon_Familial_Adenomatous_Polyposis
Gross and microscopic description of adenomatous polyps in familial adenomatous polyposis.
Video source: Pathweb
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- published: 27 Nov 2019
- views: 4888
4:01
What is Familial Adenomatous Polyposis or FAP?
From ASCO 2011 -- A discussion with Eric Charsky, disabled veteran, cancer survivor, and Founder and CEO of Volontie 1.
From ASCO 2011 -- A discussion with Eric Charsky, disabled veteran, cancer survivor, and Founder and CEO of Volontie 1.
https://wn.com/What_Is_Familial_Adenomatous_Polyposis_Or_Fap
From ASCO 2011 -- A discussion with Eric Charsky, disabled veteran, cancer survivor, and Founder and CEO of Volontie 1.
- published: 27 Jun 2011
- views: 6375
10:41
Laparoscopic Assisted Restorative Proctocolectomy for Familial Adenomatous Polyposis
Authors: Steven D. Wexner, MD, FACS, FRCS, FRCS (Ed); Jill C. Genua, MD; David A. Vivas, MD
Authors: Steven D. Wexner, MD, FACS, FRCS, FRCS (Ed); Jill C. Genua, MD; David A. Vivas, MD
https://wn.com/Laparoscopic_Assisted_Restorative_Proctocolectomy_For_Familial_Adenomatous_Polyposis
Authors: Steven D. Wexner, MD, FACS, FRCS, FRCS (Ed); Jill C. Genua, MD; David A. Vivas, MD
- published: 27 Jun 2011
- views: 2510
2:20
Familial Adenomatous Polyposis (FAP)
Watch the video to know about FAP and its associated syndromes.
Watch the video to know about FAP and its associated syndromes.
https://wn.com/Familial_Adenomatous_Polyposis_(Fap)
Watch the video to know about FAP and its associated syndromes.
- published: 04 Mar 2016
- views: 21006
1:53
Gastric adenomas and their management in familial adenomatous polyposis
Isabel Martin on "Gastric adenomas and their management in familial adenomatous polyposis"
Authors: Isabel Martin, Victorine H. Roos, Chukwuemeka Anele et al.
...
Isabel Martin on "Gastric adenomas and their management in familial adenomatous polyposis"
Authors: Isabel Martin, Victorine H. Roos, Chukwuemeka Anele et al.
Summary:
Among 726 patients with familial adenomatous polyposis (FAP), 104 (14%) had a gastric adenoma. High grade dysplasia was detected in five (5%) of these patients, while two patients had gastric cancer at initial gastric adenoma diagnosis. During follow-up, three patients were additionally diagnosed with gastric cancer. The study shows the importance of gastric endoscopic surveillance in patients with FAP.
Bibliography
Endoscopy 2021; 53: 795–801
DOI 10.1055/a-1265-2716
ISSN 0013-726X
© 2020. Thieme. All rights reserved.
Georg Thieme Verlag KG, Rüdigerstraße 14,
70469 Stuttgart, Germany
Read more here: https://doi.org/10.1055/a-1265-2716
https://wn.com/Gastric_Adenomas_And_Their_Management_In_Familial_Adenomatous_Polyposis
Isabel Martin on "Gastric adenomas and their management in familial adenomatous polyposis"
Authors: Isabel Martin, Victorine H. Roos, Chukwuemeka Anele et al.
Summary:
Among 726 patients with familial adenomatous polyposis (FAP), 104 (14%) had a gastric adenoma. High grade dysplasia was detected in five (5%) of these patients, while two patients had gastric cancer at initial gastric adenoma diagnosis. During follow-up, three patients were additionally diagnosed with gastric cancer. The study shows the importance of gastric endoscopic surveillance in patients with FAP.
Bibliography
Endoscopy 2021; 53: 795–801
DOI 10.1055/a-1265-2716
ISSN 0013-726X
© 2020. Thieme. All rights reserved.
Georg Thieme Verlag KG, Rüdigerstraße 14,
70469 Stuttgart, Germany
Read more here: https://doi.org/10.1055/a-1265-2716
- published: 27 Jul 2021
- views: 237
2:05
Attenuated- Familial Adenomatous Polyposis
Attenuated (Mild Type) Familial Adenomatous Polyposis
Colorectal cancers usually develop from polyps. If there is an adenomatous polyp, that means there is a...
Attenuated (Mild Type) Familial Adenomatous Polyposis
Colorectal cancers usually develop from polyps. If there is an adenomatous polyp, that means there is a risk of cancer. This is bad news. The good news is that removing polyps eliminates the risk of cancer.
Polyps are best diagnosed with colonoscopy and can be removed with the help of colonoscopy. If there are too many polyps that cannot be removed by colonoscopy, the large intestine including the polyps should be surgically removed.
Some genetic diseases can cause huge numbers of polyps in the large intestine. These genetic diseases pass from the parents to their children.
A genetic disease that causes more than 100 polyps in the large intestine is called familial polyposis. There is also a mild type of this disease. In the mild type, the number of polyps in the large intestine is between 20 and 100.
If measures are not taken in familial polyposis, the risk of large bowel cancer is 100%. Even in the mild form, the risk of colorectal cancer is almost 70%. In order to eliminate the risk of cancer in patients with polyposis, the large intestine should be surgically removed.
Following removal of the large intestine, a connection between the small intestine and the last part of the large intestine allows the patient to continue his life without a stoma.
In this video, images of the removed large intestine in a patient with mild-type familial polyposis are shared.
The connection between the small intestine and the last part of the large intestine ensured the patient to continue his life without an abdominal stoma.
https://wn.com/Attenuated_Familial_Adenomatous_Polyposis
Attenuated (Mild Type) Familial Adenomatous Polyposis
Colorectal cancers usually develop from polyps. If there is an adenomatous polyp, that means there is a risk of cancer. This is bad news. The good news is that removing polyps eliminates the risk of cancer.
Polyps are best diagnosed with colonoscopy and can be removed with the help of colonoscopy. If there are too many polyps that cannot be removed by colonoscopy, the large intestine including the polyps should be surgically removed.
Some genetic diseases can cause huge numbers of polyps in the large intestine. These genetic diseases pass from the parents to their children.
A genetic disease that causes more than 100 polyps in the large intestine is called familial polyposis. There is also a mild type of this disease. In the mild type, the number of polyps in the large intestine is between 20 and 100.
If measures are not taken in familial polyposis, the risk of large bowel cancer is 100%. Even in the mild form, the risk of colorectal cancer is almost 70%. In order to eliminate the risk of cancer in patients with polyposis, the large intestine should be surgically removed.
Following removal of the large intestine, a connection between the small intestine and the last part of the large intestine allows the patient to continue his life without a stoma.
In this video, images of the removed large intestine in a patient with mild-type familial polyposis are shared.
The connection between the small intestine and the last part of the large intestine ensured the patient to continue his life without an abdominal stoma.
- published: 12 Sep 2020
- views: 1027