-
Approach to Shock - types, hypovolemic, cardiogenic, distributive, anaphylaxis, pathology, treatment
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You can send me mail:
📫 PO BOX 166, Randwick Post Office, Randwick Plaza, NSW 2031, Australi...
published: 25 Jun 2022
-
Shock | Clinical Medicine
Premium Member Resources: https://www.ninjanerd.org/lecture/shock
Ninja Nerds!
During the upcoming lecture, Professor Zach Murphy will provide a comprehensive overview of Shock, including its various types, diagnostic procedures, and treatment options. The lecture will cover Hypovolemic, Obstructive, Cardiogenic, and Distributive shock, multisystem organ failure (MOF), lab findings, right heart catheterization, and medical and surgical therapies. Please show your support below!
Table of Contents:
0:00 Lab
0:07 Shock Introduction
0:58 Pathophysiology | Hypovolemic Shock
5:36 Pathophysiology | Obstructive Shock
9:46 Pathophysiology | Distributive Shock
15:49 Pathophysiology | Cardiogenic Shock
21:20 Complications | Multisystem Organ Failure (MOF)
32:41 Diagnostic Approach
37:25 Treatment
4...
published: 11 Mar 2024
-
Shock, Pathology of Different Types, Animation
(USMLE topics, cardiology) Types of circulatory shock: hypovolemic, cardiogenic, obstructive, distributive (septic, anaphylactic and neurogenic), pathophysiology, signs, symptoms, presentation and diagnosis.
Purchase a license to download a non-watermarked version of this video on AlilaMedicalMedia(dot)com
Check out our new Alila Academy - AlilaAcademy(dot)com - complete video courses with quizzes, PDFs, and downloadable images.
©Alila Medical Media. All rights reserved.
Voice by Ashley Fleming
All images/videos by Alila Medical Media are for information purposes ONLY and are NOT intended to replace professional medical advice, diagnosis or treatment. Always seek the advice of a qualified healthcare provider with any questions you may have regarding a medical condition.
Shock, also...
published: 29 Oct 2018
-
Types of Shock for Nursing | Shock NCLEX Tips Made Easy
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Understanding vari...
published: 17 Aug 2022
-
Hypovolemic Shock | Shock (Part 3)
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In this 3rd lesson, we explore and break down hypovolemic shock. Our goal is to hopefully have hypovolemic shock made easy.
First we start off covering the three main categories of causes that can lead to a state of hypovolemic shock. We then talk about the hypovolemic shock pathophysiology as well as the hypovolemic shock symptoms, treatment, and management. We cover all the bases so that you have a solid foundation for a good understanding of what is going on.
As the lessons in this series go on, we will ...
published: 28 May 2019
-
Cardiogenic Shock Nursing Management, Pathophysiology, Interventions NCLEX Review
Cardiogenic shock nursing review on the management, pathophysiology, causes, symptoms, and nursing interventions NCLEX review.
Cardiogenic shock occurs when the heart can NOT pump enough blood to meet the perfusion needs of the body. This results in low cardiac output and cell perfusion is decreased to the organs and tissues.
Cardiogenic management includes: reperfusion to the heart muscle if possible via a heart cath, administration of medications that increase cardiac output and stroke volume (vasopressors, inotropic vasopressors, vasodilators, diuretics etc.), hemodynamic monitoring, management of the respiratory system due to pulmonary edema, placement of intra-aortic balloon pump etc.
Symptoms of cardiogenic shock arise from the decreased cardiac output by the heart and decreased ...
published: 22 Dec 2018
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Anaphylactic Shock (Anaphylaxis) Treatment, Nursing Interventions, Symptoms NCLEX
Anaphylactic shock nursing review of the treatment, signs and symptoms, nursing interventions for anaphylaxis and anaphylactoid shock reactions.
Anaphylactic shock occurs when an allergen causes the mast cells or basophils to release massive amount of histamine and other mediators system-wide. This leads to a decrease in tissue perfusion to cells.
The release of histamine causes vasodilation, increased capillary permeability, bronchoconstriction, increased gastric secretions and smooth muscle contraction.
Signs and symptoms of anaphylactic shock include: difficulty breathing, inability to speak, swelling, wheezing, runny nose, watery eyes, hypotension, increased heart rate, itchy skin, diarrhea, nausea, vomiting etc.
Treatment and nursing interventions for anaphylactic shock include: r...
published: 18 Feb 2019
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THE 4 STAGES OF SHOCK
Shock is a medical emergency which can result in organ damage and death. A complex physiological response is triggered by decreased tissue perfusion. There are 4 types of shock depending on underlying cause: hypovolemic, cardiogenic, obstructive, and distributive, which encompasses anaphylactic, septic, and neurogenic shock. The 4 stages of shock are initial, compensatory, progressive, and refractory.
The initial stage may be difficult to recognize due to subtle or absent symptoms. Something has led to a decrease in tissue perfusion. Cardiac output is so low, cells experience hypoxia. Without sufficient oxygen and nutrients, tissues switch from aerobic metabolism to anaerobic metabolism. A by-product of anaerobic metabolism is lactic acid. Normally, the liver deals with lactic acid. But du...
published: 25 Apr 2023
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Shock | Types And Mechanisms of Shock | Dr Najeeb🩺
#mechanisms #drnajeeblectures #typesofshock #shock #drnajeeb
Shock | Types And Mechanisms of Shock | Dr Najeeb
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published: 23 Jun 2022
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Neurogenic Shock | Shock (Part 7)
In the 7th lesson in our series of lessons on shock, we take a look at Neurogenic Shock! This is the second shock type we discuss within the category of distributive shock.
We start of by talking about what exactly neurogenic shock is and what is happening inside the body that ultimately leads to a state of shock.
From there we talk about the signs that you would see in your patient if they were in neurogenic shock. And then finally we discuss some of the treatment modalities that you would look to implement if your patient was in this type of shock.
Neurogenic shock made easy by thoroughly explaining what is going on and helping to make sense of this very unique type of shock.
-+-+-+-+-+-
Please if you enjoyed this video or found it useful, hit the like button as this greatly helps ...
published: 10 Jun 2019
13:25
Approach to Shock - types, hypovolemic, cardiogenic, distributive, anaphylaxis, pathology, treatment
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https://you...
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https://wn.com/Approach_To_Shock_Types,_Hypovolemic,_Cardiogenic,_Distributive,_Anaphylaxis,_Pathology,_Treatment
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Daphne McLeod
You can send me mail:
📫 PO BOX 166, Randwick Post Office, Randwick Plaza, NSW 2031, Australia
- published: 25 Jun 2022
- views: 218216
40:29
Shock | Clinical Medicine
Premium Member Resources: https://www.ninjanerd.org/lecture/shock
Ninja Nerds!
During the upcoming lecture, Professor Zach Murphy will provide a comprehensive ...
Premium Member Resources: https://www.ninjanerd.org/lecture/shock
Ninja Nerds!
During the upcoming lecture, Professor Zach Murphy will provide a comprehensive overview of Shock, including its various types, diagnostic procedures, and treatment options. The lecture will cover Hypovolemic, Obstructive, Cardiogenic, and Distributive shock, multisystem organ failure (MOF), lab findings, right heart catheterization, and medical and surgical therapies. Please show your support below!
Table of Contents:
0:00 Lab
0:07 Shock Introduction
0:58 Pathophysiology | Hypovolemic Shock
5:36 Pathophysiology | Obstructive Shock
9:46 Pathophysiology | Distributive Shock
15:49 Pathophysiology | Cardiogenic Shock
21:20 Complications | Multisystem Organ Failure (MOF)
32:41 Diagnostic Approach
37:25 Treatment
40:08 Comment, Like, SUBSCRIBE!
Retired Ninja Nerd Lectures:
https://youtu.be/V-WgoUD7mIk
https://youtu.be/t_RLS6sSFrE
https://youtu.be/UbJRZTFLKdw
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#ninjanerd #cardiovascular #shock
https://wn.com/Shock_|_Clinical_Medicine
Premium Member Resources: https://www.ninjanerd.org/lecture/shock
Ninja Nerds!
During the upcoming lecture, Professor Zach Murphy will provide a comprehensive overview of Shock, including its various types, diagnostic procedures, and treatment options. The lecture will cover Hypovolemic, Obstructive, Cardiogenic, and Distributive shock, multisystem organ failure (MOF), lab findings, right heart catheterization, and medical and surgical therapies. Please show your support below!
Table of Contents:
0:00 Lab
0:07 Shock Introduction
0:58 Pathophysiology | Hypovolemic Shock
5:36 Pathophysiology | Obstructive Shock
9:46 Pathophysiology | Distributive Shock
15:49 Pathophysiology | Cardiogenic Shock
21:20 Complications | Multisystem Organ Failure (MOF)
32:41 Diagnostic Approach
37:25 Treatment
40:08 Comment, Like, SUBSCRIBE!
Retired Ninja Nerd Lectures:
https://youtu.be/V-WgoUD7mIk
https://youtu.be/t_RLS6sSFrE
https://youtu.be/UbJRZTFLKdw
Ninja Nerd Website | https://ninjanerd.org
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https://www.instagram.com/ninjanerdlectures/
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https://www.facebook.com/NinjaNerdLectures/
https://twitter.com/@NinjaNerdSci
https://discord.gg/@TAsV8BGd
#ninjanerd #cardiovascular #shock
- published: 11 Mar 2024
- views: 89320
3:55
Shock, Pathology of Different Types, Animation
(USMLE topics, cardiology) Types of circulatory shock: hypovolemic, cardiogenic, obstructive, distributive (septic, anaphylactic and neurogenic), pathophysiolog...
(USMLE topics, cardiology) Types of circulatory shock: hypovolemic, cardiogenic, obstructive, distributive (septic, anaphylactic and neurogenic), pathophysiology, signs, symptoms, presentation and diagnosis.
Purchase a license to download a non-watermarked version of this video on AlilaMedicalMedia(dot)com
Check out our new Alila Academy - AlilaAcademy(dot)com - complete video courses with quizzes, PDFs, and downloadable images.
©Alila Medical Media. All rights reserved.
Voice by Ashley Fleming
All images/videos by Alila Medical Media are for information purposes ONLY and are NOT intended to replace professional medical advice, diagnosis or treatment. Always seek the advice of a qualified healthcare provider with any questions you may have regarding a medical condition.
Shock, also called circulatory shock, is a life-threatening clinical state characterized by body-wide deficiency of blood supply, causing oxygen deprivation, buildup of waste products, and eventual organ failure if untreated.
Shock may have different causes and hence its classification into different types:
- Hypovolemic, or low volume shock happens when the circulating blood volume is severely reduced. This can be caused by:
+ External blood loss, such as after an injury,
+ Internal blood loss such as that results from a ruptured blood vessel, ruptured ectopic pregnancy, pancreatitis …
+ Or fluid loss from major burns, excessive vomiting, diarrhea or urination.
- Cardiogenic shock occurs when the heart fails to pump sufficiently. This can result from a sudden heart attack, or an end-stage development in various heart conditions.
- Obstructive shock is caused by an obstruction of blood flow in a major circulatory circuit.
- Distributive shock results from excessive dilation of blood vessels, or vasodilation, which decreases blood pressures. Distribution shock can have different causes, the most common being sepsis, anaphylaxis and damage to the central nervous system (neurogenic):
+ In sepsis, the immune system is overwhelmed by an infection that gets out of control, and responds with a systemic cytokine release that causes vasodilation and fluid leakage from capillaries.
+ In anaphylaxis, the immune system overreacts to an allergen, releasing massive amounts of histamine, which has similar effects to cytokines. Peanut allergy is a common cause of anaphylaxis.
+ Neurogenic shock typically occurs as a result of a spinal cord injury. As the autonomic nervous system is damaged, the sympathetic tone that normally keeps blood vessels constricted is lost, causing vasodilation and hypotension.
Common symptoms of shock include low blood pressures and signs of organ damage such as confusion, reduced urine output and cold, sweaty, mottled or bluish skin, although distributive shocks due to sepsis or anaphylaxis may initially produce warm or flushed skin. This is because the infection in sepsis usually comes with fever, and the allergic reaction in anaphylaxis is accompanied by hives. Distributive shocks may also differ from other types of shock by having, at least initially, normal or high cardiac output.
As the body tries to compensate for hypotension, fast heart rates and rapid breathing may be observed. Diagnosis may also be assisted by blood tests for blood lactate levels. This is because in the absence of oxygen, the body switches to an alternative way of producing cellular energy, called anaerobic metabolism, in which glucose is broken down only partially producing lactic acid. Blood tests may also indicate signs of organ damage, or infection in case of sepsis.
Shock is a medical emergency and requires immediate treatments which aim to increase blood pressures and treat the underlying cause.
https://wn.com/Shock,_Pathology_Of_Different_Types,_Animation
(USMLE topics, cardiology) Types of circulatory shock: hypovolemic, cardiogenic, obstructive, distributive (septic, anaphylactic and neurogenic), pathophysiology, signs, symptoms, presentation and diagnosis.
Purchase a license to download a non-watermarked version of this video on AlilaMedicalMedia(dot)com
Check out our new Alila Academy - AlilaAcademy(dot)com - complete video courses with quizzes, PDFs, and downloadable images.
©Alila Medical Media. All rights reserved.
Voice by Ashley Fleming
All images/videos by Alila Medical Media are for information purposes ONLY and are NOT intended to replace professional medical advice, diagnosis or treatment. Always seek the advice of a qualified healthcare provider with any questions you may have regarding a medical condition.
Shock, also called circulatory shock, is a life-threatening clinical state characterized by body-wide deficiency of blood supply, causing oxygen deprivation, buildup of waste products, and eventual organ failure if untreated.
Shock may have different causes and hence its classification into different types:
- Hypovolemic, or low volume shock happens when the circulating blood volume is severely reduced. This can be caused by:
+ External blood loss, such as after an injury,
+ Internal blood loss such as that results from a ruptured blood vessel, ruptured ectopic pregnancy, pancreatitis …
+ Or fluid loss from major burns, excessive vomiting, diarrhea or urination.
- Cardiogenic shock occurs when the heart fails to pump sufficiently. This can result from a sudden heart attack, or an end-stage development in various heart conditions.
- Obstructive shock is caused by an obstruction of blood flow in a major circulatory circuit.
- Distributive shock results from excessive dilation of blood vessels, or vasodilation, which decreases blood pressures. Distribution shock can have different causes, the most common being sepsis, anaphylaxis and damage to the central nervous system (neurogenic):
+ In sepsis, the immune system is overwhelmed by an infection that gets out of control, and responds with a systemic cytokine release that causes vasodilation and fluid leakage from capillaries.
+ In anaphylaxis, the immune system overreacts to an allergen, releasing massive amounts of histamine, which has similar effects to cytokines. Peanut allergy is a common cause of anaphylaxis.
+ Neurogenic shock typically occurs as a result of a spinal cord injury. As the autonomic nervous system is damaged, the sympathetic tone that normally keeps blood vessels constricted is lost, causing vasodilation and hypotension.
Common symptoms of shock include low blood pressures and signs of organ damage such as confusion, reduced urine output and cold, sweaty, mottled or bluish skin, although distributive shocks due to sepsis or anaphylaxis may initially produce warm or flushed skin. This is because the infection in sepsis usually comes with fever, and the allergic reaction in anaphylaxis is accompanied by hives. Distributive shocks may also differ from other types of shock by having, at least initially, normal or high cardiac output.
As the body tries to compensate for hypotension, fast heart rates and rapid breathing may be observed. Diagnosis may also be assisted by blood tests for blood lactate levels. This is because in the absence of oxygen, the body switches to an alternative way of producing cellular energy, called anaerobic metabolism, in which glucose is broken down only partially producing lactic acid. Blood tests may also indicate signs of organ damage, or infection in case of sepsis.
Shock is a medical emergency and requires immediate treatments which aim to increase blood pressures and treat the underlying cause.
- published: 29 Oct 2018
- views: 634328
22:50
Types of Shock for Nursing | Shock NCLEX Tips Made Easy
Did you know there’s only a few full length videos on our channel? The rest are trimmed down to only 20% of the content! Head to https://bit.ly/3RQpP5y for excl...
Did you know there’s only a few full length videos on our channel? The rest are trimmed down to only 20% of the content! Head to https://bit.ly/3RQpP5y for exclusive access to our FULL video library with over 1,200 in-depth videos.
See why SimpleNursing is trusted by over 1,000,000 nursing students by working smarter, NOT harder.
A SimpleNursing Membership includes:
🎥 1,200+ fun, animated videos (80% NOT on YouTube)
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For the NCLEX:
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Understanding various shock types like hypovolemic, cardiogenic, anaphylactic, and septic is critical knowledge for nursing school exams and the NCLEX.
This video details pathophysiology, causes, patient manifestations, labs, treatments, and nursing interventions to stabilize hemodynamics for each classification. Recognizing early signs of shock enables rapid, potentially life-saving treatment for compromised oxygen perfusion. Reviewing the major characteristics differentiating types of shock ensures students understand this essential emergency response topic for testing and clinical practice.
#shock #typesofshock #nursing #nclex
https://wn.com/Types_Of_Shock_For_Nursing_|_Shock_Nclex_Tips_Made_Easy
Did you know there’s only a few full length videos on our channel? The rest are trimmed down to only 20% of the content! Head to https://bit.ly/3RQpP5y for exclusive access to our FULL video library with over 1,200 in-depth videos.
See why SimpleNursing is trusted by over 1,000,000 nursing students by working smarter, NOT harder.
A SimpleNursing Membership includes:
🎥 1,200+ fun, animated videos (80% NOT on YouTube)
📚 900+ colorful study guides packed with memory tricks
🧠 3,000+ practice questions
For the NCLEX:
🎥 1,200+ fun, animated videos (80% NOT on YouTube)
📚 900+ colorful study guides packed with memory tricks
🧠 4,000+ practice questions
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❓ NGN Practice Questions
... and more!
Click the links above to try it out risk-free!
Understanding various shock types like hypovolemic, cardiogenic, anaphylactic, and septic is critical knowledge for nursing school exams and the NCLEX.
This video details pathophysiology, causes, patient manifestations, labs, treatments, and nursing interventions to stabilize hemodynamics for each classification. Recognizing early signs of shock enables rapid, potentially life-saving treatment for compromised oxygen perfusion. Reviewing the major characteristics differentiating types of shock ensures students understand this essential emergency response topic for testing and clinical practice.
#shock #typesofshock #nursing #nclex
- published: 17 Aug 2022
- views: 756800
12:29
Hypovolemic Shock | Shock (Part 3)
‼️🎓 Want to earn CE credits for watching these videos? Join ICU Advantage Academy. 👉🏼 https://adv.icu/academy
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In this 3rd lesson, we explore and break down hypovolemic shock. Our goal is to hopefully have hypovolemic shock made easy.
First we start off covering the three main categories of causes that can lead to a state of hypovolemic shock. We then talk about the hypovolemic shock pathophysiology as well as the hypovolemic shock symptoms, treatment, and management. We cover all the bases so that you have a solid foundation for a good understanding of what is going on.
As the lessons in this series go on, we will take a deep dive in to each type, or cause, of shock and break it down much in the same way. After covering them all, we will conclude with a lesson covering how to determine which type of shock your patient is in.
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Also make sure and subscribe and hit the bell notification to stay up to date on our latest videos!
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https://www.facebook.com/icuadvantage/
-+-+-+-+-+-
You can check out the first lesson in this series by clicking here: https://youtu.be/n77OH16wztk
For the next lesson in this series covering cardiogenic shock, click here: https://youtu.be/Ctf8I_5wMSQ
You can also check out the playlist for this series of lesson on shock here: https://youtu.be/n77OH16wztk
Don't forget to check out our 6-part series of lessons covering Hemodynamics here: https://youtu.be/5EchkYvRkxs
https://wn.com/Hypovolemic_Shock_|_Shock_(Part_3)
‼️🎓 Want to earn CE credits for watching these videos? Join ICU Advantage Academy. 👉🏼 https://adv.icu/academy
💰🤑 10% off Critical Care Academy (CCRN Review): 👉🏼 https://adv.icu/cca (USE CODE "icuadv10")
💰🤑 10% off EACH Month @ My Mastery Nursing membership: 👉🏼 https://adv.icu/mastery
In this 3rd lesson, we explore and break down hypovolemic shock. Our goal is to hopefully have hypovolemic shock made easy.
First we start off covering the three main categories of causes that can lead to a state of hypovolemic shock. We then talk about the hypovolemic shock pathophysiology as well as the hypovolemic shock symptoms, treatment, and management. We cover all the bases so that you have a solid foundation for a good understanding of what is going on.
As the lessons in this series go on, we will take a deep dive in to each type, or cause, of shock and break it down much in the same way. After covering them all, we will conclude with a lesson covering how to determine which type of shock your patient is in.
-+-+-+-+-+-
Please if you enjoyed this video or found it useful, hit the like button as this greatly helps our channel out!
Also make sure and subscribe and hit the bell notification to stay up to date on our latest videos!
Finally make sure to make your way over to our Facebook page and give us a like and shout out over there!
https://www.facebook.com/icuadvantage/
-+-+-+-+-+-
You can check out the first lesson in this series by clicking here: https://youtu.be/n77OH16wztk
For the next lesson in this series covering cardiogenic shock, click here: https://youtu.be/Ctf8I_5wMSQ
You can also check out the playlist for this series of lesson on shock here: https://youtu.be/n77OH16wztk
Don't forget to check out our 6-part series of lessons covering Hemodynamics here: https://youtu.be/5EchkYvRkxs
- published: 28 May 2019
- views: 174443
32:23
Cardiogenic Shock Nursing Management, Pathophysiology, Interventions NCLEX Review
Cardiogenic shock nursing review on the management, pathophysiology, causes, symptoms, and nursing interventions NCLEX review.
Cardiogenic shock occurs when th...
Cardiogenic shock nursing review on the management, pathophysiology, causes, symptoms, and nursing interventions NCLEX review.
Cardiogenic shock occurs when the heart can NOT pump enough blood to meet the perfusion needs of the body. This results in low cardiac output and cell perfusion is decreased to the organs and tissues.
Cardiogenic management includes: reperfusion to the heart muscle if possible via a heart cath, administration of medications that increase cardiac output and stroke volume (vasopressors, inotropic vasopressors, vasodilators, diuretics etc.), hemodynamic monitoring, management of the respiratory system due to pulmonary edema, placement of intra-aortic balloon pump etc.
Symptoms of cardiogenic shock arise from the decreased cardiac output by the heart and decreased tissue perfusion. Signs and symptoms of cardiogenic shock include: back flow of blood into the lungs and right side of the heart leading to pulmonary edema, jugular venous distention, dyspnea, increased respiratory rate, tachycardia, pale, cool skin, low urinary output, mental status changes, increased pulmonary artery wedge pressure and central venous pressures.
Nursing interventions for cardiogenic shock include: assessing for adequate tissue perfusion, maintaining hemodynamic monitoring, titrating cardiac medications, etc.
Quiz: https://www.registerednursern.com/cardiogenic-shock-nclex-questions/
Notes: https://www.registerednursern.com/cardiogenic-shock-nclex-review/
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#shock #cardiogenicshock #shocktypes #shocknursing
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https://wn.com/Cardiogenic_Shock_Nursing_Management,_Pathophysiology,_Interventions_Nclex_Review
Cardiogenic shock nursing review on the management, pathophysiology, causes, symptoms, and nursing interventions NCLEX review.
Cardiogenic shock occurs when the heart can NOT pump enough blood to meet the perfusion needs of the body. This results in low cardiac output and cell perfusion is decreased to the organs and tissues.
Cardiogenic management includes: reperfusion to the heart muscle if possible via a heart cath, administration of medications that increase cardiac output and stroke volume (vasopressors, inotropic vasopressors, vasodilators, diuretics etc.), hemodynamic monitoring, management of the respiratory system due to pulmonary edema, placement of intra-aortic balloon pump etc.
Symptoms of cardiogenic shock arise from the decreased cardiac output by the heart and decreased tissue perfusion. Signs and symptoms of cardiogenic shock include: back flow of blood into the lungs and right side of the heart leading to pulmonary edema, jugular venous distention, dyspnea, increased respiratory rate, tachycardia, pale, cool skin, low urinary output, mental status changes, increased pulmonary artery wedge pressure and central venous pressures.
Nursing interventions for cardiogenic shock include: assessing for adequate tissue perfusion, maintaining hemodynamic monitoring, titrating cardiac medications, etc.
Quiz: https://www.registerednursern.com/cardiogenic-shock-nclex-questions/
Notes: https://www.registerednursern.com/cardiogenic-shock-nclex-review/
Shock Series: https://www.youtube.com/playlist?list=PLQrdx7rRsKfWj2TbPcyAcNCilffYLe3NK
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Diabetes Health Managment: https://www.youtube.com/playlist?list=PLQrdx7rRsKfXtEx17D7zC1efmWIX-iIs9
- published: 22 Dec 2018
- views: 901021
19:32
Anaphylactic Shock (Anaphylaxis) Treatment, Nursing Interventions, Symptoms NCLEX
Anaphylactic shock nursing review of the treatment, signs and symptoms, nursing interventions for anaphylaxis and anaphylactoid shock reactions.
Anaphylactic s...
Anaphylactic shock nursing review of the treatment, signs and symptoms, nursing interventions for anaphylaxis and anaphylactoid shock reactions.
Anaphylactic shock occurs when an allergen causes the mast cells or basophils to release massive amount of histamine and other mediators system-wide. This leads to a decrease in tissue perfusion to cells.
The release of histamine causes vasodilation, increased capillary permeability, bronchoconstriction, increased gastric secretions and smooth muscle contraction.
Signs and symptoms of anaphylactic shock include: difficulty breathing, inability to speak, swelling, wheezing, runny nose, watery eyes, hypotension, increased heart rate, itchy skin, diarrhea, nausea, vomiting etc.
Treatment and nursing interventions for anaphylactic shock include: removing the allergen, administering epinephrine, placing the patient in the supine position with the leg elevated, education etc.
#anaphylacticshock #shock #anaphylaxis
Quiz: https://www.registerednursern.com/anaphylactic-shock-nclex-questions/
Notes: https://www.registerednursern.com/anaphylactic-shock-nclex-review/
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https://wn.com/Anaphylactic_Shock_(Anaphylaxis)_Treatment,_Nursing_Interventions,_Symptoms_Nclex
Anaphylactic shock nursing review of the treatment, signs and symptoms, nursing interventions for anaphylaxis and anaphylactoid shock reactions.
Anaphylactic shock occurs when an allergen causes the mast cells or basophils to release massive amount of histamine and other mediators system-wide. This leads to a decrease in tissue perfusion to cells.
The release of histamine causes vasodilation, increased capillary permeability, bronchoconstriction, increased gastric secretions and smooth muscle contraction.
Signs and symptoms of anaphylactic shock include: difficulty breathing, inability to speak, swelling, wheezing, runny nose, watery eyes, hypotension, increased heart rate, itchy skin, diarrhea, nausea, vomiting etc.
Treatment and nursing interventions for anaphylactic shock include: removing the allergen, administering epinephrine, placing the patient in the supine position with the leg elevated, education etc.
#anaphylacticshock #shock #anaphylaxis
Quiz: https://www.registerednursern.com/anaphylactic-shock-nclex-questions/
Notes: https://www.registerednursern.com/anaphylactic-shock-nclex-review/
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Diabetes Health Managment: https://www.youtube.com/playlist?list=PLQrdx7rRsKfXtEx17D7zC1efmWIX-iIs9
- published: 18 Feb 2019
- views: 387982
7:35
THE 4 STAGES OF SHOCK
Shock is a medical emergency which can result in organ damage and death. A complex physiological response is triggered by decreased tissue perfusion. There are ...
Shock is a medical emergency which can result in organ damage and death. A complex physiological response is triggered by decreased tissue perfusion. There are 4 types of shock depending on underlying cause: hypovolemic, cardiogenic, obstructive, and distributive, which encompasses anaphylactic, septic, and neurogenic shock. The 4 stages of shock are initial, compensatory, progressive, and refractory.
The initial stage may be difficult to recognize due to subtle or absent symptoms. Something has led to a decrease in tissue perfusion. Cardiac output is so low, cells experience hypoxia. Without sufficient oxygen and nutrients, tissues switch from aerobic metabolism to anaerobic metabolism. A by-product of anaerobic metabolism is lactic acid. Normally, the liver deals with lactic acid. But during shock, the liver's not working optimally. Lactic acid builds up in the bloodstream, dropping blood pH and causing acidosis.
Next is the compensatory stage. Compensatory mechanisms try to release substances that increase cardiac output or increase blood volume to increase tissue perfusion. The body attempts to increase cardiac output and blood pressure with the SNS and RAS.
Baroreceptors sense a drop in blood pressure and stimulate the SNS. The SNS releases catecholamines epinephrine and norepinephrine. These catecholamines cause vasoconstriction, increased blood pressure, and increased heart rate. Less blood goes to non-vital organs, more blood is routed to vital ones. The drop in blood pressure results in decreased capillary hydrostatic pressure. This triggers an increase in venous blood by shifting fluid from the interstitial compartment to the intravascular compartment.
Baroreceptors also stimulate the vagus nerve, which stimulates the release of antidiuretic hormone from the posterior pituitary gland, preventing water from leaving the kidneys and thus increasing blood volume.
The RAS kicks into gear. A reduction in blood flow to the kidneys triggers them to convert prorenin in blood to renin. Renin is released into circulation, where it converts angiotensinogen from the liver, to angiotensin I. With the help of ACE, angiotensin I is converted to angiotensin II. Angiotensin II is a vasoconstrictive peptide which narrows both arteries and veins. Angiotensin II also triggers the release of aldosterone from the adrenal cortex, which makes the kidneys retain more sodium and water and increase potassium excretion - more retention of water in the bloodstream and increased blood pressure.
Compensatory mechanisms increase cardiac output and blood volume. But some compromises are made. Perfusion is decreased to the GI tract, so it slows. There is decreased perfusion to the skin, which makes it cold and clammy (exception to this is if the person is in septic shock).
Decreased perfusion results in parts of the lungs not getting perfusion, which means no gas exchange in those parts. There is a mismatch between ventilation and perfusion and blood oxygen levels decrease so the person hyperventilates.
The body can only maintain compensatory mechanisms for a limited time.
During the progressive stage, compensatory mechanisms have failed. They no longer maintain adequate tissue perfusion, which leads to worsening tissue damage. The body progresses towards MODS. No more compensation means low cardiac output and low tissue perfusion. Cells do not receive oxygen, succumbing to cell hypoxic injury.
Capillary permeability increases - this is key to the pathology of this stage. The barrier between intravascular and interstitial space is broken down. Fluid and protein are drawn into the interstitial space, resulting in major edema. This depletes blood volume, decreases cardiac output and tissue perfusion.
What happens in various organs? When the brain doesn't receive adequate perfusion, there is a major mental status change. Heart cells begin to die, including those of the electrical conduction system. This results in cardiac dysrhythmias. In the lungs, ARDS develops. Increased capillary permeability in alveolar sacs, the site of gas exchange, results in their collapse. This results in fluid in the lungs, lower oxygen levels, high respiratory rate, and respiratory failure. The person requires intubation and mechanical ventilation in order to keep breathing. In the GI tract, ulcers form as the cells that protect the gut’s lining from its own acid stop working. This results in massive gastrointestinal bleeding. Liver’s cells are also dying. Since the liver produces most clotting factors, clotting doesn’t work well.
There is also disseminated intravascular coagulation. Small clots form in vessels, further blocking blood flow to organs. These clots deplete platelets and clotting factors, resulting in massive and uncontrolled bleeding.
The fourth and final stage of shock is the refractory stage, characterized by poor tissue perfusion, hypotension, and organ failure. Despite aggressive resuscitation efforts, the person is unlikely to survive.
https://wn.com/The_4_Stages_Of_Shock
Shock is a medical emergency which can result in organ damage and death. A complex physiological response is triggered by decreased tissue perfusion. There are 4 types of shock depending on underlying cause: hypovolemic, cardiogenic, obstructive, and distributive, which encompasses anaphylactic, septic, and neurogenic shock. The 4 stages of shock are initial, compensatory, progressive, and refractory.
The initial stage may be difficult to recognize due to subtle or absent symptoms. Something has led to a decrease in tissue perfusion. Cardiac output is so low, cells experience hypoxia. Without sufficient oxygen and nutrients, tissues switch from aerobic metabolism to anaerobic metabolism. A by-product of anaerobic metabolism is lactic acid. Normally, the liver deals with lactic acid. But during shock, the liver's not working optimally. Lactic acid builds up in the bloodstream, dropping blood pH and causing acidosis.
Next is the compensatory stage. Compensatory mechanisms try to release substances that increase cardiac output or increase blood volume to increase tissue perfusion. The body attempts to increase cardiac output and blood pressure with the SNS and RAS.
Baroreceptors sense a drop in blood pressure and stimulate the SNS. The SNS releases catecholamines epinephrine and norepinephrine. These catecholamines cause vasoconstriction, increased blood pressure, and increased heart rate. Less blood goes to non-vital organs, more blood is routed to vital ones. The drop in blood pressure results in decreased capillary hydrostatic pressure. This triggers an increase in venous blood by shifting fluid from the interstitial compartment to the intravascular compartment.
Baroreceptors also stimulate the vagus nerve, which stimulates the release of antidiuretic hormone from the posterior pituitary gland, preventing water from leaving the kidneys and thus increasing blood volume.
The RAS kicks into gear. A reduction in blood flow to the kidneys triggers them to convert prorenin in blood to renin. Renin is released into circulation, where it converts angiotensinogen from the liver, to angiotensin I. With the help of ACE, angiotensin I is converted to angiotensin II. Angiotensin II is a vasoconstrictive peptide which narrows both arteries and veins. Angiotensin II also triggers the release of aldosterone from the adrenal cortex, which makes the kidneys retain more sodium and water and increase potassium excretion - more retention of water in the bloodstream and increased blood pressure.
Compensatory mechanisms increase cardiac output and blood volume. But some compromises are made. Perfusion is decreased to the GI tract, so it slows. There is decreased perfusion to the skin, which makes it cold and clammy (exception to this is if the person is in septic shock).
Decreased perfusion results in parts of the lungs not getting perfusion, which means no gas exchange in those parts. There is a mismatch between ventilation and perfusion and blood oxygen levels decrease so the person hyperventilates.
The body can only maintain compensatory mechanisms for a limited time.
During the progressive stage, compensatory mechanisms have failed. They no longer maintain adequate tissue perfusion, which leads to worsening tissue damage. The body progresses towards MODS. No more compensation means low cardiac output and low tissue perfusion. Cells do not receive oxygen, succumbing to cell hypoxic injury.
Capillary permeability increases - this is key to the pathology of this stage. The barrier between intravascular and interstitial space is broken down. Fluid and protein are drawn into the interstitial space, resulting in major edema. This depletes blood volume, decreases cardiac output and tissue perfusion.
What happens in various organs? When the brain doesn't receive adequate perfusion, there is a major mental status change. Heart cells begin to die, including those of the electrical conduction system. This results in cardiac dysrhythmias. In the lungs, ARDS develops. Increased capillary permeability in alveolar sacs, the site of gas exchange, results in their collapse. This results in fluid in the lungs, lower oxygen levels, high respiratory rate, and respiratory failure. The person requires intubation and mechanical ventilation in order to keep breathing. In the GI tract, ulcers form as the cells that protect the gut’s lining from its own acid stop working. This results in massive gastrointestinal bleeding. Liver’s cells are also dying. Since the liver produces most clotting factors, clotting doesn’t work well.
There is also disseminated intravascular coagulation. Small clots form in vessels, further blocking blood flow to organs. These clots deplete platelets and clotting factors, resulting in massive and uncontrolled bleeding.
The fourth and final stage of shock is the refractory stage, characterized by poor tissue perfusion, hypotension, and organ failure. Despite aggressive resuscitation efforts, the person is unlikely to survive.
- published: 25 Apr 2023
- views: 44914
3:07:41
Shock | Types And Mechanisms of Shock | Dr Najeeb🩺
#mechanisms #drnajeeblectures #typesofshock #shock #drnajeeb
Shock | Types And Mechanisms of Shock | Dr Najeeb
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▬▬▬▬▬▬▬▬▬▬ Contents of this video ▬▬▬▬▬▬▬▬▬▬
0:00 Types, Mechanisms & Stages of Shock
Definitions of different types of shocks;
HYPOVOLEMIC SHOCK
CARDIOGENIC SHOCK
DISTRIBUTIVE SHOCK & its 3 types:
1-Anaphylactic Shock
2-Neurogenic Shock
3-Septic Shock
22:22 OBSTRUCTIVE SHOCK
CAUSES of HYPOVOLEMIC SHOCK
1-HEMORRHAGE:
External Hemorrhage
Internal Hemorrhage
2-LOSS OF PLASMA:
Exfoliative Dermatitis
3-LOSS OF FLUIDS & ELECTROLYTES:
Diarrhea & Vomiting
Polyuria: e.g., Diabetic Ketoacidosis
Excessive Sweating
3rd Spacing; Ascites, Bowel Obstruction, Acute Pancreatitis,
36:20 CARDIOGENIC SHOCK
1-ARRHYTHMIAS:
Tachyarrhythmia & Bradyarrhythmia
2-TRUE PUMP FAILURE:
Massive Myocardial Infarction
Cardiomyopathies
CARDIOMYOPATHIES
3-SEVERE VALVULAR DYSFUNCTION:
Valvular Regurgitation; like Mitral Valve Regurgitation, Aortic Valve Regurgitation.
4-RUPTURE OF VENTRICULAR SEPTUM or FREE WALL
53:09 DISTRIBUTIVE SHOCK:
1-Neurogenic Shock:
Vasomotor Center Failure/Depression
2-Anaphylactic Shock
3-Septic Shock
1:02:35 Acute Adrenal Insufficiency
1:21:21 OBSTRUCTIVE SHOCK:
1-Cardiac Tamponade
2-Tension Pneumothorax
3-Massive Pulmonary Embolism
4-Atrial Myxoma
5-Left Atrial Mural Thrombus
1:15:20 Syncope; Vasovagal shock.
Outline of STAGES OF SHOCK:
1-Non-Progressive Compensated Stage
2-Progressive Stage of Shock
3-Refractory Shock/Irreversible Shock
4-NON-PROGRESSIVE COMPENSATED STAGE SHOCK; in detail:
1-Sympathetic Autonomous Nervous System (SANS):
Chemoreceptors, Baroreceptors, Vasomotor Center, Vasoconstriction & Arterioloconstriction
Cardiac Output (CO) & Systolic Blood Pressure (SBP), Total Peripheral Resistance (TPR) & Diastolic Blood Pressure (DBP)
Compensatory mechanisms try to stabilize the blood pressure.
2-Renin-Angiotensin-Aldosterone Axis System (RAAA):
Reduced renal perfusion, Renin release, Angiotensin-1, Angiotensinogen (ATG), Angiotensin-2 (AT-2)
Why more Renin is released?
Arterioloconstriction leads to increased TPR; DBP stabilized.
AT-2 act on Adrenal Cortex; ZG will release Aldosterone; salt & water retained; increased blood volume;
SBP stabilized.
3-ADH(Vasopressin
4-REVERSE STRESS-RELAXATION of Circulatory System
5-FLUID SHIFT (Interstitial to Vascular)
1:52:07 PROGRESSIVE STAGE OF SHOCK:
Compensatory Mechanisms are exhausted and are failing; deterioration starts.
Fight b/w Vasoconstrictor & Vasodilator mechanisms.
"Peripheral tissues will become resistant to the compensatory vasoconstrictor mechanisms & start vasodilating".
Endothelial Cells are injured; NO is released which acts as Vasodilator and cardio depressant.
Lactic Acidosis
Disseminated Intravascular Coagulation (DIC)
2:06:45 REFRACTORY IRREVERSIBLE STAGE:
"Cells & tissues start committing suicide".
1-Na+K+ ATPases are not working
2-Lysosomal Membrane breaks down
3-ATP into Adenosine; raised uric acid in the blood.
2:16:36 Post mortem findings in these patients.
SEPTIC SHOCK & its types:
1-Bacteremia
2-Septicemia
3-Endotoxemia
Differences b/w in Gram +ve & Gram -ve bacteria.
Differences b/w Endotoxins & Exotoxins.
4-Pyemia
5-Empyema
6-Abscess
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Shock | Types And Mechanisms of Shock | Dr Najeeb
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▬▬▬▬▬▬▬▬▬▬ Contents of this video ▬▬▬▬▬▬▬▬▬▬
0:00 Types, Mechanisms & Stages of Shock
Definitions of different types of shocks;
HYPOVOLEMIC SHOCK
CARDIOGENIC SHOCK
DISTRIBUTIVE SHOCK & its 3 types:
1-Anaphylactic Shock
2-Neurogenic Shock
3-Septic Shock
22:22 OBSTRUCTIVE SHOCK
CAUSES of HYPOVOLEMIC SHOCK
1-HEMORRHAGE:
External Hemorrhage
Internal Hemorrhage
2-LOSS OF PLASMA:
Exfoliative Dermatitis
3-LOSS OF FLUIDS & ELECTROLYTES:
Diarrhea & Vomiting
Polyuria: e.g., Diabetic Ketoacidosis
Excessive Sweating
3rd Spacing; Ascites, Bowel Obstruction, Acute Pancreatitis,
36:20 CARDIOGENIC SHOCK
1-ARRHYTHMIAS:
Tachyarrhythmia & Bradyarrhythmia
2-TRUE PUMP FAILURE:
Massive Myocardial Infarction
Cardiomyopathies
CARDIOMYOPATHIES
3-SEVERE VALVULAR DYSFUNCTION:
Valvular Regurgitation; like Mitral Valve Regurgitation, Aortic Valve Regurgitation.
4-RUPTURE OF VENTRICULAR SEPTUM or FREE WALL
53:09 DISTRIBUTIVE SHOCK:
1-Neurogenic Shock:
Vasomotor Center Failure/Depression
2-Anaphylactic Shock
3-Septic Shock
1:02:35 Acute Adrenal Insufficiency
1:21:21 OBSTRUCTIVE SHOCK:
1-Cardiac Tamponade
2-Tension Pneumothorax
3-Massive Pulmonary Embolism
4-Atrial Myxoma
5-Left Atrial Mural Thrombus
1:15:20 Syncope; Vasovagal shock.
Outline of STAGES OF SHOCK:
1-Non-Progressive Compensated Stage
2-Progressive Stage of Shock
3-Refractory Shock/Irreversible Shock
4-NON-PROGRESSIVE COMPENSATED STAGE SHOCK; in detail:
1-Sympathetic Autonomous Nervous System (SANS):
Chemoreceptors, Baroreceptors, Vasomotor Center, Vasoconstriction & Arterioloconstriction
Cardiac Output (CO) & Systolic Blood Pressure (SBP), Total Peripheral Resistance (TPR) & Diastolic Blood Pressure (DBP)
Compensatory mechanisms try to stabilize the blood pressure.
2-Renin-Angiotensin-Aldosterone Axis System (RAAA):
Reduced renal perfusion, Renin release, Angiotensin-1, Angiotensinogen (ATG), Angiotensin-2 (AT-2)
Why more Renin is released?
Arterioloconstriction leads to increased TPR; DBP stabilized.
AT-2 act on Adrenal Cortex; ZG will release Aldosterone; salt & water retained; increased blood volume;
SBP stabilized.
3-ADH(Vasopressin
4-REVERSE STRESS-RELAXATION of Circulatory System
5-FLUID SHIFT (Interstitial to Vascular)
1:52:07 PROGRESSIVE STAGE OF SHOCK:
Compensatory Mechanisms are exhausted and are failing; deterioration starts.
Fight b/w Vasoconstrictor & Vasodilator mechanisms.
"Peripheral tissues will become resistant to the compensatory vasoconstrictor mechanisms & start vasodilating".
Endothelial Cells are injured; NO is released which acts as Vasodilator and cardio depressant.
Lactic Acidosis
Disseminated Intravascular Coagulation (DIC)
2:06:45 REFRACTORY IRREVERSIBLE STAGE:
"Cells & tissues start committing suicide".
1-Na+K+ ATPases are not working
2-Lysosomal Membrane breaks down
3-ATP into Adenosine; raised uric acid in the blood.
2:16:36 Post mortem findings in these patients.
SEPTIC SHOCK & its types:
1-Bacteremia
2-Septicemia
3-Endotoxemia
Differences b/w in Gram +ve & Gram -ve bacteria.
Differences b/w Endotoxins & Exotoxins.
4-Pyemia
5-Empyema
6-Abscess
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- published: 23 Jun 2022
- views: 500013
10:51
Neurogenic Shock | Shock (Part 7)
In the 7th lesson in our series of lessons on shock, we take a look at Neurogenic Shock! This is the second shock type we discuss within the category of distri...
In the 7th lesson in our series of lessons on shock, we take a look at Neurogenic Shock! This is the second shock type we discuss within the category of distributive shock.
We start of by talking about what exactly neurogenic shock is and what is happening inside the body that ultimately leads to a state of shock.
From there we talk about the signs that you would see in your patient if they were in neurogenic shock. And then finally we discuss some of the treatment modalities that you would look to implement if your patient was in this type of shock.
Neurogenic shock made easy by thoroughly explaining what is going on and helping to make sense of this very unique type of shock.
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For the next lesson in this series covering septic shock, click here: **COMING SOON**
You can also check out the playlist for this series of lesson on shock here: https://youtu.be/n77OH16wztk
Also check out our other popular series of lessons on Hemodynamic Principals: https://youtu.be/5EchkYvRkxs
https://wn.com/Neurogenic_Shock_|_Shock_(Part_7)
In the 7th lesson in our series of lessons on shock, we take a look at Neurogenic Shock! This is the second shock type we discuss within the category of distributive shock.
We start of by talking about what exactly neurogenic shock is and what is happening inside the body that ultimately leads to a state of shock.
From there we talk about the signs that you would see in your patient if they were in neurogenic shock. And then finally we discuss some of the treatment modalities that you would look to implement if your patient was in this type of shock.
Neurogenic shock made easy by thoroughly explaining what is going on and helping to make sense of this very unique type of shock.
-+-+-+-+-+-
Please if you enjoyed this video or found it useful, hit the like button as this greatly helps our channel out!
Also make sure and subscribe and hit the bell notification to stay up to date on our latest videos!
Facebook: https://www.facebook.com/icuadvantage/
-+-+-+-+-+-
For the next lesson in this series covering septic shock, click here: **COMING SOON**
You can also check out the playlist for this series of lesson on shock here: https://youtu.be/n77OH16wztk
Also check out our other popular series of lessons on Hemodynamic Principals: https://youtu.be/5EchkYvRkxs
- published: 10 Jun 2019
- views: 65629