EULAR recommendations for the treatment of systemic sclerosis: 2023 update
Annals of the Rheumatic Diseases (ARD)
is a leading rheumatology journal from EULAR, publishing original research, reviews and recommendations
Impact Factor: 20.3
Citescore: 35
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Latest EULAR Recommendations
EULAR recommendations for the involvement of patient research partners in rheumatology research: 2023 update
EULAR recommendations for the management of psoriatic arthritis with pharmacological therapies: 2023 update
2023 EULAR recommendations on imaging in diagnosis and management of crystal-induced arthropathies in clinical practice
Authors
Annals of the Rheumatic Diseases will be published by Elsevier from January 2025. All new manuscript submissions should be submitted online at https://www.editorialmanager.com/ards/default2.aspx.
Latest Articles
Recommendation:
Expert consensus recommendations for the diagnosis and treatment of chronic non-bacterial osteitis (CNO) in adults28 November 2024
Rheumatoid arthritis:
Ivarmacitinib, a selective Janus kinase 1 inhibitor, in patients with moderate-to-severe active rheumatoid arthritis and inadequate response to conventional synthetic DMARDs: results from a phase III randomised clinical trial27 November 2024
Correction:
Correction: Olokizumab plus methotrexate: safety and efficacy over 106 weeks of treatment27 November 2024
Systemic lupus erythematosus:
Pharmacodynamics of the S1P1 receptor modulator cenerimod in a phase 2b randomised clinical trial in patients with moderate to severe SLE24 November 2024
Recommendations
The EULAR Standing Committees regularly establish and publish recommendations for the treatment of various rheumatic disorders. Each of these recommendations is developed by a group of specialists in their field.
All EULAR recommendations are free to access.
Each recommendation also has an accompanying lay summary, which is also free to access.
Video Abstract
m6A RNA methylation controls salivary gland epithelial cell function and has a protective role in Sjögren’s disease
Conclusions: Impairment of m6A modifications in SGEC in response to inflammatory triggers favour the formation of dsRNA, potentially amplifying the interferon loop and contributing to SjD pathogenesis.
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