SH3BP5
المظهر
SH3BP5 (SH3 domain binding protein 5) هوَ بروتين يُشَفر بواسطة جين SH3BP5 في الإنسان.[1][2][3]
الوظيفة
[عدل]هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
الأهمية السريرية
[عدل]هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
المراجع
[عدل]- ^ Yamadori T؛ Baba Y؛ Matsushita M؛ Hashimoto S؛ Kurosaki M؛ Kurosaki T؛ Kishimoto T؛ Tsukada S (يونيو 1999). "Bruton's tyrosine kinase activity is negatively regulated by Sab, the Btk-SH3 domain-binding protein". Proc Natl Acad Sci U S A. ج. 96 ع. 11: 6341–6. Bibcode:1999PNAS...96.6341Y. DOI:10.1073/pnas.96.11.6341. PMC:26883. PMID:10339589.
- ^ "Entrez Gene: SH3BP5 SH3-domain binding protein 5 (BTK-associated)". مؤرشف من الأصل في 2010-12-05.
- ^ Matsushita M؛ Yamadori T؛ Kato S؛ Takemoto Y؛ Inazawa J؛ Baba Y؛ Hashimoto S؛ Sekine S؛ Arai S؛ Kunikata T؛ Kurimoto M؛ Kishimoto T؛ Tsukada S (يونيو 1998). "Identification and characterization of a novel SH3-domain binding protein, Sab, which preferentially associates with Bruton's tyrosine kinase (BtK)". Biochem Biophys Res Commun. ج. 245 ع. 2: 337–43. DOI:10.1006/bbrc.1998.8420. PMID:9571151.
قراءة متعمقة
[عدل]- Baba Y، Matsushita M، Matsuda Y، وآخرون (2000). "Assignment of SH3BP5/Sh3bp5 encoding sab, an SH3 domain-binding protein which preferentially associates with Bruton's tyrosine kinase, to human chromosome 1q43 and mouse chromosome 14B by in situ hybridization". Cytogenet. Cell Genet. ج. 87 ع. 3–4: 221–2. DOI:10.1159/000015430. PMID:10702676.
- Wiltshire C، Matsushita M، Tsukada S، وآخرون (2002). "A new c-Jun N-terminal kinase (JNK)-interacting protein, Sab (SH3BP5), associates with mitochondria". Biochem. J. ج. 367 ع. Pt 3: 577–85. DOI:10.1042/BJ20020553. PMC:1222945. PMID:12167088.
- Strausberg RL، Feingold EA، Grouse LH، وآخرون (2003). "Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences". Proc. Natl. Acad. Sci. U.S.A. ج. 99 ع. 26: 16899–903. Bibcode:2002PNAS...9916899M. DOI:10.1073/pnas.242603899. PMC:139241. PMID:12477932.
- Ota T، Suzuki Y، Nishikawa T، وآخرون (2004). "Complete sequencing and characterization of 21,243 full-length human cDNAs". Nat. Genet. ج. 36 ع. 1: 40–5. DOI:10.1038/ng1285. PMID:14702039.
- Brandenberger R، Wei H، Zhang S، وآخرون (2005). "Transcriptome characterization elucidates signaling networks that control human ES cell growth and differentiation". Nat. Biotechnol. ج. 22 ع. 6: 707–16. DOI:10.1038/nbt971. PMID:15146197.
- Court NW؛ Kuo I؛ Quigley O؛ Bogoyevitch MA (2004). "Phosphorylation of the mitochondrial protein Sab by stress-activated protein kinase 3". Biochem. Biophys. Res. Commun. ج. 319 ع. 1: 130–7. DOI:10.1016/j.bbrc.2004.04.148. PMID:15158451.
- Gerhard DS، Wagner L، Feingold EA، وآخرون (2004). "The status, quality, and expansion of the NIH full-length cDNA project: the Mammalian Gene Collection (MGC)". Genome Res. ج. 14 ع. 10B: 2121–7. DOI:10.1101/gr.2596504. PMC:528928. PMID:15489334.
- Wiltshire C؛ Gillespie DA؛ May GH (2005). "Sab (SH3BP5), a novel mitochondria-localized JNK-interacting protein". Biochem. Soc. Trans. ج. 32 ع. Pt 6: 1075–7. DOI:10.1042/BST0321075. PMID:15506969.
- Rual JF، Venkatesan K، Hao T، وآخرون (2005). "Towards a proteome-scale map of the human protein-protein interaction network". Nature. ج. 437 ع. 7062: 1173–8. Bibcode:2005Natur.437.1173R. DOI:10.1038/nature04209. PMID:16189514.
- Beausoleil SA، Villén J، Gerber SA، وآخرون (2006). "A probability-based approach for high-throughput protein phosphorylation analysis and site localization". Nat. Biotechnol. ج. 24 ع. 10: 1285–92. DOI:10.1038/nbt1240. PMID:16964243.